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Above: Manchester, UK

For those who picked up on the first post I hope that you took the time to go on to Sarah Knowles second and third posts on the topic. If not, then here are the links…

‘Science vs The Gap’

https://datapatientaction.wordpress.com/2018/01/24/three-cheers-for-implementation-science-2-science-vs-the-gap/

and, ‘You Make Me Want to be a Better Researcher’

https://datapatientaction.wordpress.com/2018/01/24/three-cheers-for-implementation-science-3-you-make-me-want-to-be-a-better-researcher/

Worth a read. I was lucky to meet Sarah and other members of the Greater Manchester NIHR CLAHRC on a visit last year. Sarah is a proper implementation scientist – I am not!

These posts are good for the uninitiated (i.e. those people who are thinking ‘what is implementation science anyway?’) and those wondering why I’m posting about it.

My favourite is Cheer #3 – probably the least well recognised and understood but for me the most important because it emphasises the two-way connection that should exist between clinical research and implementation science. It’s what takes implementation science from being an academic curiosity to a ‘thing’ that clinician scientists benefit from understanding and engaging with.

 

The last couple of years have been busy and seen change. In the true spirit of ‘generalism’ I now have three jobs.

My practice,

My public practice, and

A university position…

The latter, as the Clinical Director, of The Australian Centre for Health Service Innovation (AusHSI) is perhaps the biggest diversion from my work as a cardiologist but sits squarely within the direction of my research work and has offered fascinating opportunities I had never expected. Read more about AusHSI here, and visit the AusHSI Blog here.

In addition to this has been growing responsibilities with the Cardiac Society (CSANZ) and editorial duties at Heart, Lung and Circulation.

Hence the difficulty finding time for the blog…

Having read some other comparable blog posts of late, and written at least one piece for another site I’m encouraged to try and resuscitate this site.

The nature of the posts will need to change. Time is never plentiful and it is a challenge at the best of the times to be able to spend it writing original material.

So the long, rambling posts will likely be a thing of the past to be replace more than likely with links to material elsewhere that I have either contributed to or fit broadly into themes or areas of interest.

But fundamentally the original goals and intentions of The Last General Cardiologist will remain (and I’m yet to be convinced that the title isn’t appropriate…

Cross blogged from our Emergency Cardiology Site…

The Emergency Cardiology Group

There has been lots of media interest in our paper ‘Validation of high-sensitivity troponin I in a 2-h diagnostic strategy to assess 30-day outcomes in emergency-department patients with possible acute coronary syndrome‘ that went online in in the Journal of the American College of Cardiology in the last few days.

Those in Queensland may have heard reference to the study on the ABC Radio news this morning and Louise Cullen has been interviewed today by ABC Radio National and Channel Nine.

We may have lost out to Black Caviar on the Channel Nine News though…

A copy of the press release from the Queensland Government is available here: RBWH media release – improved testing for potential heart attack patient.

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This sort of thing has been bugging me for ages…

Old CardioBrief

Don’t believe the the hype! That’s the cardinal rule to obey when reading health news. “Breakthroughs” and “cures” are rare, and should always be viewed with caution and skepticism.

This week was a great example. Last Sunday, the New York Times, the major networks, and a host of other media outlets (including this one) reported on a paper in  Nature Medicine about the discovery of a novel and potentially significant pathway linking red meat to heart disease. Briefly, the research suggested that carnitine, which is found naturally in high concentrations in red meat, can lead to atherosclerosis when it is converted by gut bacteria to a chemical called TMAO. Almost immediately I received a lot of comment from experts who raised serious questions about the research. Then today, a separate study was published with an entirely different perspective on carnitine. Although the two studies don’t directly contradict each other, they…

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